Hello and help

2007-11-30 19:44:17

Hello,I'm Ros from Berks.uk.I've only recently found you so please
bear with me.I've had cp for about five years but only recently been
diagnosed,it seems that info and support is very ltd. in the uk.I
have the usual (it seems)pain and have been referred to a pain clinic
wich I'm waiting on,but recently have been having some strange
symptoms(for me).I'e been having episodes(usually about 3pm)where I
become shaky,sweaty,weak and can feel my heart racing.Ialso become
irritable and have problems concentrating.I feel as though I'm hungry
but have no appetite.I have been having a snack and sitting quietly
and after half an hour or so it goes off.Does this ring any bells
with anyone?I feel it must be conected to cp.but cant find any
information or anyone else whos had the same sort of thing.I've read
that diabetes is a complicatoin but this would seem to be
different.I've read that glucon is excreted by the pancreasand the
doctor has said I have 5% approx function, could it be that I am not
producing sufficient?I would welcome any suggestions or comments.Many
thanks and I hope you all have an angel on your shoulder tonight.Ros

Hosted Chat, 4/7/2004, 7:00 pm

2007-11-30 17:22:40

Reminder Reminder from the Calendar of ThePancreatitisPlace
Hosted Chat
Wednesday April 7, 2004
7:00 pm - 9:00 pm
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ReeAnn@...

Re: I am new TO SALLY

2007-11-30 12:47:42

hi Sally,
you have come to the right place...i was diagnosed 3 yrs ago, im a
30 yr old..have 2 small kids...lost in this world just as you are
now..didnt know why i was the one to have this disease and was
trying to find someone with the same thing..someone that would
understand what i am going through...
well sally u have found the perfect place.. a family that will
always be here for you..through everything ( a shoulder to cry on,
someone to just complain to, or just to have someone put a smile on
your face).
take care,ur friend brenda
have a pain free day

Links about Pancreatitis+++++Kimber

2007-11-30 01:27:50

Sally,
glad I could be of some help. Unfortunately, for many of us, this
disease and it's pain is here to stay. I'm one of those. There is a lot
of information out there regarding pancreatitis, but it is still not
fully understood by the doctors. This is one of the reason it is so hard
to diagnose and treat.
Kimber
--
Kimber Allen
Vallejo, CA
hominid2@...
Note: All advice given is personal opinion, not equal to that of a licensed
physician or health care professional.

Low Fat Sheppards Pie

2007-11-30 00:31:17

Rita,
This is a recipe that I make at the house. I got it from my mom, but
she used ground beef instead of turkey. I changed it up a little, but
it is still good. Turkey does not have the fat that ground beef has.
And my family cannot tell the difference.This is a very good recipe
(so my family tells me) and it is very low in fat.Hope you enjoy.
God Bless
Denise~S.C.
Caregivers Moderator
denise@...
http://www.thepancreatitisplace.org/

Re: hello Rita HAMBURGER CORNMEAL SHEPHERD'S PIE

2007-11-29 18:45:21

Rita,
Here is a good recipe for Shepards Pie. It is a little different than
the regular Pie with the mashed potatoes, but I thought I would send
it to you. Some more recipes are coming for you.
Denise~S.C.
Caregivers Moderator
denise@...
http://www.thepancreatitisplace.org/

Pancreatic site

2007-11-29 11:52:38

04/06/04,
Kimber, this is Laura Kerste from Toledo, Ohio and I haven't
been on the site the last few days. Pancreas is flaring up and may
end up in the hospital. But I did see your message on the site you
gave Sally.
I put it in my favorites, because it is late at this moment.
But the first chance I can go to the site I will. It sounds very
interesting.
Thank You for posting this. It might just help alot of our
members.
Thanks, Laura Kerste

Pancreatic Diseases

2007-11-29 09:05:42

http://www.labtestsonline.org/understanding/conditions/pancreatic_dise
ases.html
The Pancreas
Pancreatic Diseases
What is the pancreas?
The pancreas is a narrow, flat organ about six inches long, with a
head, middle, and tail section. It is located below the liver,
between the stomach and the spine, and its head section connects to
the duodenum (first part of the small intestine). Inside the
pancreas, small ducts (tubes) feed fluids produced by the pancreas
into the pancreatic duct. This larger duct carries the fluids down
the length of the pancreas, from the tail to the head, and into the
duodenum. The common bile duct also runs through the head section of
the pancreas, carrying bile from the liver and gall bladder into the
small intestine.
The pancreas consists of two kinds of tissues:
Exocrine -- which make powerful enzymes to digest fats, proteins, and
carbohydrates. The enzymes normally are created and carried to the
duodenum in an inactive form, then activated as needed. Exocrine
tissue also makes bicarbonates that work to neutralize stomach acids.
Endocrine -- which produce the hormones insulin and glucagon and
release them into the blood stream. These hormones regulate glucose
transport into the body's cells and are crucial for energy
production.
Acute Pancreatitis
The most common cause of acute pancreatitis is blockage of the
pancreatic duct by a gallstone. Secretions can back up in the
pancreas and cause permanent damage in just a few hours. They also
can circulate to other body organs, causing shock and organ failure.
Acute pancreatitis can be life-threatening.
Chronic Pancreatitis
Chronic pancreatitis, associated most often with gall bladder disease
and alcoholism, can cause painful attacks over a number of years and
lead to other problems, such as pancreatic insufficiency (see below),
bacterial infections, and type 2 diabetes.
Diabetes
Type 1 diabetes involves destruction of pancreatic beta cells (which
produce insulin) and a dependence on insulin; type 2 involves loss of
some beta cell function and insulin resistance. Diabetes affects many
other body organs, especially the kidneys.
Cystic Fibrosis
CF is an inherited genetic disorder that disrupts chloride transport
at the cellular level. This causes mucous plugs that block pancreatic
enzymes from reaching the intestines and leads to digestive problems.
Pancreatic Cancer
Cancer of the pancreas is the fourth leading cause of cancer death in
the United States, killing about 29,000 people a year. Risks include
smoking, age, gender (more common in men), chronic pancreatitis, and
exposure to some industrial chemicals. Most (95%) pancreatic cancers
are adenocarcinomas, developing in the exocrine tissues. Pancreatic
cancer is very difficult to detect in the early stages because
symptoms are either absent or nonspecific: abdominal pain, nausea,
loss of appetite, and sometimes jaundice. Only about 10% of the
cancers are still contained within the pancreas at the time of
diagnosis
Pancreatic Insufficiency
Pancreatic insufficiency is the inability of the pancreas to produce
and/or transport enough digestive enzymes to break down food in the
intestine and allow its absorption. It typically occurs as a result
of chronic pancreatic damage - damage that may be caused by a variety
of conditions. It is most frequently associated with cystic fibrosis
in children and with chronic pancreatitis in adults; it is less
frequently but sometimes associated with pancreatic cancer.

Pancreatic Functions

2007-11-28 18:56:10

http://www.gastroresource.com/GITextbook/en/chapter12/12-3-pr.htm
Pancreatic Functions
3.1 Secretory Studies
It is easy to diagnose pancreatic insufficiency in the presence of
the clinical triad of pancreatic calcification, diabetes and
steatorrhea. Most pancreatic diseases, however, remain clinically
silent until approximately 90% of the gland is destroyed. Lipase
secretion appears to decrease earlier than trypsin secretion; hence,
steatorrhea appears earlier than azotorrhea in patients suffering
from pancreatic disease. Earlier recognition of pancreatic
dysfunction may improve the management of the patient's disease and
his or her quality of life.
Pancreatic function tests may be divided into two main groups: direct
(duodenal intubation) and indirect (Table 1).
TABLE 1. Exocrine pancreatic function

Re: I am new For Sally

2007-11-28 18:34:16

Hey Sally,
Glad to hear from you again. Yes, just click on the links to the
left. It shouldnt take you into cyber space---I konw about that--
happens to me too!!! Sorry that you are having so much pain. I do not
have pancreatitis. My husband, Robert has been dealing with it for 15
years. I am his caregiver. I love TPP and love meeting and talking to
everybody. I hope you find it 'home' here for you.Our prayers are
with you and your family.
God Bless
Denise~S.C.
Caregivers Moderator
denise@...
http://www.thepancreatitisplace.org/

Amylase Tests

2007-11-28 07:43:45

http://www.ahealthyme.com/topic/topic100586423;jsessionid=AZOPJGFINLS3
ACTYAITC4EQ?_requestid=45072
Amylase Tests
Definition
Amylase is a digestive enzyme made primarily by the pancreas and
salivary glands. Enzymes are substances made and used by the body to
trigger specific chemical reactions. The primary function of the
enzyme amylase is to break down starches in food so that they can be
used by the body. Amylase testing is usually done to determine the
cause of sudden abdominal pain.
Purpose
Amylase testing is performed to diagnose a number of diseases that
elevate amylase levels. Pancreatitis, for example, is the most common
reason for a high amylase level. When the pancreas is inflamed,
amylase escapes from the pancreas into the blood. Within six to 48
hours after the pain begins, amylase levels in the blood start to
rise. Levels will stay high for several days before gradually
returning to normal.
There are other causes of increased amylase. An ulcer that erodes
tissue from the stomach and goes into the pancreas will cause amylase
to spill into the blood. During a mumps infection, amylase from the
inflamed salivary glands increases. Amylase is also found in the
liver, fallopian tubes, and small intestine; inflammation of these
tissues also increases levels. Gall bladder disease, tumors of the
lung or ovaries, alcohol poisoning, ruptured aortic aneurysm, and
intestinal strangulation or perforation can also cause unusually high
amylase levels.
Precautions
This is not a screening test for future pancreatic disease.
Description
Amylase testing is done on both blood and urine. The laboratory may
use any of several testing methods that involve mixing the blood or
urine sample with a substance with which amylase is known to react.
By measuring the end-product or the reaction time, technicians can
calculate the amount of amylase present in the sample. More
sophisticated methods separately measure the amylase made by the
pancreas and the amylase made by the salivary glands.
Urine testing is a better long-term monitor of amylase levels. The
kidneys quickly move extra amylase from the blood into the urine.
Urine levels rise six to 10 hours after blood levels and stay high
longer. Urine is usually collected throughout a 2- or 24-hour time
period. Results are usually available the same day.
Preparation
In most cases, no special preparation is necessary for a person
undergoing an amylase blood test. Patients taking longer term urine
amylase tests will be given a container and instructions for
collecting the urine at home. The urine should be refrigerated until
it is brought to the laboratory.
Aftercare
Discomfort or bruising may occur at the puncture site or the person
may feel dizzy or faint. Pressure to the puncture site until the
bleeding stops reduces bruising. Applying warm packs to the puncture
site relieves discomfort.
Normal results
Normal results vary based on the laboratory and the method used.
Abnormal results
Eight out of ten persons with acute pancreatitis will have high
amylase levels, up to four times the normal level. Other causes of
increased amylase, such as mumps, kidney failure, pregnancy occurring
in the abdomen but outside the uterus (ectopic pregnancy), certain
tumors, a penetrating ulcer, certain complications of diabetes, and
advanced pancreatic cancer, are further investigated based on the
person's symptoms, medical history, and the results of other tests.
In kidney disease, the kidneys are not as efficient at removing
amylase from the blood. Amylase rises in the blood, but stays at
normal levels in the urine.
People with macroamylasia have large clumps of amylase in their
blood. These clumps are too large to move through the kidney, so they
stay in the blood. Amylase levels in the blood will be high; levels
in the urine will be low.
Amylase levels may be low in severe liver disease (including
hepatitis), conditions in which the pancreas fails to secrete enough
enzyme for proper digestions (pancreatic insufficiency), when toxic
materials build up in the blood during pregnancy (pre-eclampsia),
following burns, in thyroid disorders, and in advanced cystic
fibrosis. Some medications can raise or lower levels.
Key Terms:
Amylase
A digestive enzyme made primarily by the pancreas and salivary
glands.
Enzyme
A substance made and used by the body to trigger specific chemical
reactions.
Pancreatitis
Inflammation of the pancreas.

Pancreas test

2007-11-28 04:50:04

http://www.pancreas.org/patients/patients_ptest.html
Pancreas test
There are several tests that are commonly used to test the pancreas
for abnormalities in structure or function. Genetic tesing provides
information on risk factors and possible causes. The structure
testing includes CT scan (as shown on the HOME page), ERCP, EUS and
MRCP. For information on these tests, click on your choice below:
CT Scan
ERCP
EUS
MRCP
Function testing is more difficult to perform and more difficult to
interpret. These test are divided into direct testing (the "tubed"
secretin test) and indirect tests such as fecal elastase. Most of the
above tests are available at major centers that specialize in
pancreatic disease.

Blood Amylase

2007-11-28 04:10:11

http://www.mdadvice.com/library/test/medtest42.html
Blood Amylase
Blood Amylase
Category: Enzymes.
Subcategory: Pancreatic enzymes.
Material studied: Blood.
Estimated cost of test: $6.00.
Patient time for test: 5-10 minutes.
Reliability of test results: Good.
Available as home self-test? No.
BEFORE THE TEST
Purpose of test:
Diagnoses acute pancreatitis*.
Distinguishes between acute pancreatitis* and other causes of
abdominal pain that require immediate surgery.
Evaluates possible injury to pancreas caused by direct blows or
surgery.
Where is test performed?
Commercial laboratory, hospital, doctor's office.
Who performs test?
Lab technician, nurse, doctor.
Risks and precautions:
If tourniquet is applied on the arm too long (over 1 minute), it may
cause an inaccurate test result. Request another sample to be
collected to ensure accuracy.
Patient preparation:
Activity--No changes necessary.
Diet--Do not drink alcohol for 24 hours before the test.
Medicines--Inform the person performing the test if you have recently
taken any medications listed under Taking these drugs may affect test
results. You may be asked not to take this medication before the
test.
Disrobing--None required. Roll up sleeve only.
THE TEST
Sensory factors:
Touching--You will feel mild discomfort when the needle is inserted
into the vein or when the lancet pricks a finger, heel or ear lobe.
Seeing--You will see the technician, nurse or doctor, the basket or
tray to hold the equipment, the needles, syringes, collecting tubes
and bandages.
Feeling--Some degree of apprehension or fear is normal and should be
expected. Discomfort disappears when the test is finished.
Other senses (taste, smell, hearing)--Not affected.
Equipment used:
Needles, syringes and heparinized* collecting tubes. Sterile,
disposable equipment prevents contamination or spread of infection.
There is no risk of becoming infected with the hepatitis virus, AIDS
virus or any other infecting germ.
Description of test:
Technician, doctor or nurse applies a tourniquet or blood-pressure
cuff to the upper arm if blood is collected from a vein.
Skin over the vein to be stuck is cleaned with alcohol or other
antiseptic on a piece of cotton.
When blood is drawn from a vein, the operator feels the vein to be
used then punctures both the skin and vein in one quick stroke. The
needle used is a sterile, disposable needle attached to a sterile,
disposable syringe.
Operator withdraws the needle and transfers sample from the
collecting syringe into sterile tubes (identified with your name)
before sending samples to the laboratory for analysis. Tubes are
treated with an anti-coagulant chemical to prevent clotting.
If blood is collected from a finger, heel or ear lobe, skin over the
selected site is cleaned with an antiseptic. The operator quickly
pierces the skin to a shallow depth, using a sterile, disposable
metal lancet. The drop or two of blood produced is collected into a
capillary pipette.
AFTER THE TEST
Immediate post-test care:
Apply pressure to the puncture site with cotton provided by the
laboratory.
If a vein has been punctured, raise your entire arm over your head
while applying pressure.
Some discoloration, soreness or swelling may develop at the
venepuncture site. This responds well to moist, warm compresses
applied every 2 to 4 hours.
Activity after test:
Resume taking any medications that were withheld before the test.
Return to pretest activities right away.
Use warm compresses if blood collects under the puncture site or if
the area becomes tender, red or painful.
Time before test results available:
Test requires only a few minutes in the laboratory. Time before
results are reported to the doctor or patient varies from a few
minutes to a few days.
TEST RESULTS
Test values:
Test results are determined by radio-immunoassay*.
Normal values:
Levels range from 60 to 180 Somogyi units/dl.
What HIGH or INCREASED may indicate:
Acute pancreatitis*.
Acute salivary-gland disease*.
Obstruction of ampulla of Vater*.
Obstruction of common bile duct.
Pancreatic cancer.
Pancreatic injury from perforated peptic ulcer*.
What LOW or DECREASED may indicate:
Chronic pancreatitis*.
Cirrhosis*.
Hepatitis*.
Pancreatic cancer.
Toxemia* of pregnancy.
Taking these drugs may affect test results:
Aminosalicylic acid.
Asparginase.
Azathioprine.
Cholinergics.
Codeine.
Corticosteroids.
Cyproheptadine.
Dexamethasone.
Ethacrynic acid.
Furosemide.
Meperidine.
Mercaptopurine.
Methacholine.
Morphine.
Oral contraceptives.
Phenformin.
Rifampin.
Sulfasalazine.
Thiazides.
Triamcinolone.
Other factors that may affect test results:
Alcohol.
Recent surgery in areas close to the pancreas.
Perforated ulcer.
Perforated intestine.
Abscess*.
Spasm of the sphincter of Oddi* or macroamylasemia*.
Coughing, sneezing or talking near an open collection tube. Saliva
contains amylase.

Re: I am new++++Denise

2007-11-27 19:56:58

Denise
Thank you for your welcome. I have has so many people write to me
since I came here. I thought no one would write to me. You said
something about the links. Do I just click on them? Will it take me
out of here and get me lost in cyber space? I do that very often.I
have my galbader taken out last October 27 of last year.And I have
hurt eversince.Thank you for wanting to help me. Are you sick too? I
hope everybody don't hurt like I do. It hurts so bad sometimes you
think you are gonna die. Last weekend I thought I was going to have
to go to the hospital. The doctor had gave me some medicine for the
pain and I took it and after many hours , I could stand the pain, but
then I was sore. Do you know why I was sore? It felt like somebody
beat me in my belly with a stick.
Sally

Re: Links about Pancreatitis+++++Kimber

2007-11-27 13:31:22

Kimber
Oh Thank you for that website. I went to it first and I must have
typed it in wrong cause it took me to another world, but I finally
found it. Boy it tells you alot. I didn't know what the pancreas
did , but I am finding out things, I don't fell so stupid anymore.My
mom crys when I hurt real bad. She thinks it will go away like a bad
cold or something, but I am finding out that it will not go away. I
AM glad that I have found you guys, My mom says thank you too.She has
a coping machine hooked up to the computer and I copied some stuff
for her to read. The computer hurts her eyes.
Sally

Important News Regarding Pain Management!!

2007-11-27 03:10:14

As just reported on Medscape:
Revised Pain Management Guidelines Target Inadequate Pain Control:
A Newsmaker Interview With James N. Thompson, MD
Laurie Barclay, MD
April 5, 2004 Editor's Note: Recognizing that undertreatment of
pain is a public health priority, the Federation of State Medical
Boards (FSMB) of the United States, Inc., is recommending a revision
of guidelines stating that undertreatment of pain, like
overtreatment, is a practice violation.
A panel of medical board representatives, experts in pain management
and addiction medicine, and state and federal government
representatives convened by the FSMB recently reviewed suggested
revisions to the Model Guidelines for the Use of Controlled
Substances for the Treatment of Pain. Inspired by new insights in
pain management, especially regarding the undertreatment of pain, the
revised guidelines will be submitted to the FSMB's House of Delegates
in May for consideration as policy. Since the original guidelines
were released in 1998, more than 300,000 copies have been distributed
nationally and adopted in whole or in part by 22 state medical
boards.
How should the revised guidelines shape current practice in pain
management? To find out, Medscape's Laurie Barclay interviewed James
N. Thompson, MD, president and chief executive officer of the
Federation of State Medical Boards in Dallas, Texas.
For those who belon to Medscape, here's the link to the entire
article:
http://www.medscape.com/viewarticle/472893?mpid=27081
It is imperative that patients apprise their physicians of this
change.
-Ellen

Re: I am new +++Ruthie

2007-11-26 20:24:02

Ruthie
WOW - 10 years. That is a long time. This is not like a cold that
will go away. Man, Will I EVER get better?What is going to happen to
me? The doctor only took some blood and talked to me. Beside the
galbadder being took out- that is all he did. I get sick to my
stomach when I eat food. Mom says that I should eat soup or yougart.
But I hurt when I eat that!!! Its not hard food, so why do I still
hurt? Thank you for writing back to me, I don't want you to think I
am stupid or anything, Its just that no body around me knows anything
about this. I am not saying that I am glad that you have it too, I am
just glad that I have finally found someone that knows something
about it. Thank you so much.
Sally

After A Pancreas Or Islet Cell Transplant

2007-11-26 20:07:34

http://www.nmh.org/services/transplant/pancreas_after.html
Continuing Medical Care
Close monitoring for complications is essential immediately
following transplant surgery. We will want to see you in the
transplant offices three times each week for the first 4 to 6 weeks
after surgery. These follow-up visits are similar to pre-surgery
visits. First we will check your vital signs and draw blood. After
the lab results are back, usually about one hour, we will examine
you, review your lab results, and discuss your medications.
For each visit to the transplant office, it is important to:
Bring your pill sheet so we can be sure you are taking the right
medications in the right amounts.
Remember not to take your cyclosporin or Prograf dose until after
your blood is drawn.
Since you may still have an intravenous line, a bladder catheter or
an abdominal drainage tube when you are discharged, you may need
nursing care in your home. The transplant team can help you arrange
for visiting nurses through Northwestern Memorial Home Health Care.
Depending on your home situation and medical condition on discharge,
the transplant team may also suggest you consider staying for a time
in the Galter Carriage House apartments, which are connected to the
hospital.
The frequency of your office visits will decrease, usually after the
first 3 months. It may also be possible to receive some of the tests
and care from a local provider, depending on your location and
condition.
Possible Complications
As with any surgery, some complications may arise. Many transplant
recipients do not experience any complications. Because most
complications can be treated successfully if detected early, it is
important to be aware of signs and symptoms.
Rejection occurs when your immune system tries to attack the
transplanted pancreas and/or kidney because it recognizes them as
being "foreign". You will be taking medication to prevent rejection
for the rest of your life. In spite of the medications, rejection
episodes occur in about 25% of patients, typically between 3 to 5
weeks after transplant.
Virtually all rejection attempts can be successfully treated through
special antirejection medication. You may need to be admitted to the
hospital during a rejection episode so we can best adjust your
medications. Because you usually will not be aware that you are
having a rejection episode, we will be testing your blood and urine
regularly to detect rejection early.
If you experience complete rejection, you will have a loss of
pancreas function. Rejection is not fatal. You will need to receive
insulin injections again and you may go back on the list to receive
another transplant.
Infection is most likely immediately after surgery, when you will be
taking high doses of antirejection medication, which make you more
susceptible to illness and infection. Symptoms of infection might
include fever, fatigue, aching joints, and changes in your white
blood cell count. You will probably be given antibiotics for the
early stages of your recovery to prevent infection. You may also want
to be careful about situations in which you are exposed to many
contagious people, particularly children. Once your antirejection
medications are decreased to a maintenance level, you will be less
vulnerable to infection.
Vascular Thrombosis is the formation of a blood clot in either the
artery or vein of the transplanted pancreas. Thrombosis occurs rarely
and is of concern only during the first few days after transplant.
However, it does result in surgical removal of the transplanted
pancreas. Symptoms include a sudden increase of blood glucose and
pain over the transplanted pancreas. To prevent thrombosis, we
administer medications to thin the blood for the first few days after
surgery.
Dehydration may occur because the pancreas excretes large amounts of
fluid through the urine. Dehydration can be prevented by drinking
fluids and increasing salt intake.
Inflammation may occur in the bladder, pancreas, and urethra due to
irritation by pancreatic enzymes. Symptoms may include burning during
urination, abdominal pain, bloody urine, and fever. Treatment usually
consists of inserting a catheter into the bladder until the
inflammation subsides. This inflammation occurs only in patients
whose pancreas is bladder drained.
Medications
Your medications are one of the most important components of your
recovery. You will begin taking medications immediately after your
surgery and, although the doses will gradually decrease, you will be
taking some of them for the rest of your life. Every patient will
have a different combination of medications and will experience the
medication differently. For all patients, it is essential that you
take all medications as prescribed and notify the transplant team
right away if you experience side effects.
Neoral (Cyclosporine) is an antirejection medication. Possible side
effects include hair growth, hand tremors, gum swelling, higher blood
pressure, increase in cholesterol level and altered kidney function.
Most side effects decrease as the dose is decreased.
Prograf (Tacrolimus) is a newer antirejection medication which may
be given in place of cyclosporine. It is taken twice a day, 12 hours
apart. Possible side effects include altered kidney function,
increased blood glucose levels, tremors, stomach upset and increased
blood pressure.
Cellcept (mycophenolate mofetil) is an antirejection medication that
may be used instead of Imuran. Possible side effects include mild
diarrhea or nausea.
Thymoglobulin is an antirejection vaccine which is given
intravenously after surgery for 7 to 14 days. It may also be used to
treat a rejection episode. Side effects include fever, chills, and
aching for the first 2 or 3 days.
Rapamune is a newer medication that prevents rejection by blocking
your immune system from making a type of white blood cell. Side
effects may include increased cholesterol level, anemia, rash, joint
pain, and diarrhea.
Bactrim or Septra(Trimethoprim, Sulfamethoxazole) are antibiotics
taken for one year following transplant to help prevent urinary tract
infections. Possible side effects include lowered white blood count,
sensitivity to the sun, and altered kidney function.
Zovirax ( Acyclovir) and Gancyclovir (DHPG) are antiviral
medications used to prevent and treat viral infections that are
common after transplant. Most patients receive these medications for
the first 3 to 4 months after transplant. Other than possibly
lowering your white blood count, there are few side effects.
Sodium bicarbonate is given to replace the bicarbonate that is
secreted in the urine from the transplanted pancreas. Multiple pills
may be necessary to replenish the bicarbonate adequately. Side
effects include stomach distention and belching.
The combined effect of all antirejection medications makes you more
susceptible to skin cancers. You should use a sunscreen with SPF of
15 or higher, and limit the time you are exposed to direct sunlight.
Hats and long-sleeved shirts are also helpful.
While you are still in the hospital, you will receive a pill sheet
listing the many medications you will be taking. Your pill sheet will
help you keep track of the medicines and doses you should be taking
each day. The pill sheet will also become an important part of each
visit with the doctor, as you review your reaction and side effects
for each medication and as changes are made in the medications and
doses.
Do not stop taking or adjust the doses on any of your medications
without your doctor's permission. The most common cause of rejection
is reducing or stopping antirejection medications. If you are having
difficult side effects, discuss these with the transplant team so
they can adjust the medications for you. If you are finding the cost
of the medications too difficult, discuss this issue with the social
worker or the financial liaison on the transplant team.
UPDATED: 07-May-2001
NORTHWESTERN MEMORIAL HOSPITAL
251 EAST HURON STREET
CHICAGO, IL 60611
312.926.2000

Re: I am new++++Gary

2007-11-26 16:29:34

Gary,
Hey, thank you for writing to me too. Do you hurt too? I had a nice
lady--Ruthie write to me. She has hurt for 10 years. Man that is a
long time. I hope you have not hurt that long. This is so nice to
meet people that know why I hurt. I am so happy to find you people ,
and its not everyday that I can say that I am happy about somehting.
Sally

After Your Islet Transplant

2007-11-26 04:56:47

http://www.med.ualberta.ca/islet/edu/after.htm
Now what?
Following discharge the care of the islet cells becomes your
responsibility
You will require immunosuppressive drugs for as long as the islet
cells are functioning. If you fail to take the drugs your body will
get rid of the islet cells by a process of rejection.
You will need to monitor your blood glucose very carefully. Initially
you will require insulin. Your insulin requirements will change with
each islet cell transplant, and the transplant team will help you to
make the adjustments to them. It is important to remember that the
islet cells will take some time to settle into their new home in your
liver. We do not want to stress them during this time so we will ask
you to stay within a specific range of glucose so as not to overwork
them. We call this period a time of engrafting.
This is similar to when you plant a crop. Not every seed that you put
in the ground will grow into a plant. However, if you prepare your
soil, and feed and water the seeds, you increase your chance for a
good harvest. So it is with the islet cell transplant: if you take
good care in this initial time period, you have a better chance of
good islet cell function. This means sticking to your diet and a
healthy balanced lifestyle.
What is rejection?
Rejection is the body's natural defense against foreign cells or
particles (e.g. bacteria and viruses). Your immune system will
identify that your transplanted islet cells are not part of your
natural biological system and will then activate a process to destroy
them.
How does the immune system work?
Every cell in your body has a protein on its surface that we call an
antigen. Some cells have a lot of these proteins while some have only
a few. These antigens act as nametags so that the immune system,
which protects your body from invaders, can identify the good and the
bad cells. Your immune system has special cells called white blood
cells, which can be subdivided into two categories. The first
category is those which identify an invading cell. They attach to the
foreign cell and send signals to alert the second class of white
blood cells which then recognize the first white blood cell and the
foreign cell and specialize in destroying these foreign cells. Once
your immune system has fought off an invader it will create a group
of cells that have memories of that invader so that it can react more
quickly to it in the future.
This provides us with immunity. As a general example, when you have a
flu shot, a live (but weak) influenza (flu) vaccine is injected into
the body and the immune system will fight it off and therefore
recognize it more quickly in the future. This system protects you
from viruses, bacteria, fungus and cancer.
How do we prevent your body from destroying the islet cells?
We use medications to interfere with parts of the immune system. We
suppress your immune system just enough that you do not reject your
islet cells. These medications are called immunosuppressants. We
currently are using Tacrolimus (Prograf), Sirolimus (Rapamune) and
Daclizumab (Zenapax). These drugs work together to have a three-
pronged attack on the immune system to stop it from being activated.
Other drugs or antibodies may be used to prevent rejection depending
on results of future studies.
We must monitor blood levels of your immunosuppressive drugs closely
so that you do not reject your islet cells or have too much of these
drugs in your system. Once you are at a stable level your monitoring
will be decreased.
How to Manage Your Medications
Right drug: Check the bottle carefully to ensure the medication is
the correct drug.
Learn both names of your drugs (e.g. Sirolimus is also known as
Rapamycin). Do not memorize colors and shapes of pills instead of
learning the names, as the appearance of the drugs can vary between
manufacturers.
Right dose: Know the dosage of your medications and always check the
medication labels to ensure you are taking the right dose.
When you pick up prescriptions from the pharmacy, double-check the
label, as the pill dosage may be different from what you had last
time.
Right time: Medications need to be taken at consistent times because
you never want to open a window of opportunity for rejection.
When a drug is taken it is absorbed and there is an initial time of
high levels in the blood; then the levels decrease as the body uses
the drug and breaks it down. The timing of medications helps to
maintain appropriate levels in your body.
Right way: Take medications according to the recommendations, e.g.,
with food or on an empty stomach.
Travel: Take an extra supply of your medications, especially
immunosuppressants.
Take medications in hand luggage when travelling.
Storage: Store medications in a dry place at room temperature unless
told to refrigerate.
Do not leave medications in the car to freeze or overheat.
When camping, use an appropriate place to store drugs to prevent
extremes of temperature.
Pharmacy: Use the same pharmacy for all prescriptions (except
Tacrolimus and Sirolimus; they are only available at the University
Hospital Pharmacy) so that the pharmacist can help you avoid
interactions by knowing you and your medications
Use your pharmacist as a resource for your medications; he/she is
your last line of defense in ensuring that your medications are safe
and will not interact.
Interactions: Over-the-counter drugs must be taken with guidance and
caution as many drugs interact with your immunosuppressants. Contact
us or speak to your pharmacist for guidance.
Common Side Effects of all Immunosuppressant Medications
Every medication causes alterations in your body. Some of these
alterations are the reason that you take the medication. However,
each medication can cause undesirable changes known as side effects,
which can vary in their seriousness and intensity. We are providing
you with information about all the possible side effects that we are
aware of. You will not get all the side effects that are listed for
each drug. You may not experience any side effects. However, it is
important that you know what to expect should they occur and how to
manage them. In addition, it is important to note that you may
develop some side effects that are not yet recognized this is
research
Decreased ability to fight infection
Good handwashing is the best prevention.
Use common sense about visiting people with infections.
Get an annual flu shot.
Practice safe sex: have yourself and your partner tested for any
sexually transmitted diseases and use condoms.
Report symptoms of infection right away.
Decreased ability to fight cancer
Use SPF 15 sunblock when in the sun, wear a hat, avoid sunbathing,
and avoid the hottest time of the day (between ten and two).
Annual mammograms and prostate exams if you are over forty.
Breast and testicular self-examination is important for early
detection.
Have any unusual skin growths examined right away.
Any cancer may ultimately lead to death.
Tacrolimus (Prograf) Tacrolimus acts directly on white blood cells to
reduce their ability to identify foreign cells.
How to take
Must be taken twelve hours apart, 8 a.m. and 8 p.m., during initial
post-transplant phase; may change by thirty minutes per day to fit
personal routines.
Take consistently with or without food. Some patients need food to
avoid nausea.
Never take with grapefruit juice or grapefruit as there is an
interaction; read labels of multi-juice products.
Drug can be obtained only at the University of Alberta Hospital
Pharmacy and Foothills Hospital Pharmacy.
Make sure you have an adequate supply; out-of-town patients can have
supplies shipped if necessary.
On blood work days, always take Tacrolimus after blood is drawn; you
will need to bring the medication to the lab with you.
Side effects Renal effects Tacrolimus is hard on the kidneys
Drink three litres of water daily to help protect them
Water is best; juices increase sugars and calories; coffee and tea
make you urinate more
Try lemon or lime in water; keeping a sports bottle of water at your
side all day long helps increase water intake
Hypertension May increase your blood pressure (BP)
You may need to take daily resting BP measurements in the morning and
evening
Keep a record on the monitoring sheet provided and bring it to clinic
Neurotoxicity Headaches, tremors, and confusion can occur
Tremors will improve with time and as doses are reduced
If having severe headaches, report the problem to your transplant
team
Confusion usually improves over time
Skin changes Oiliness and acne are commonly reported
Keep your skin and hair clean
Gastrointestinal Diarrhea, nausea and vomiting can be a problem
Take your medications with food if stomach upset is a problem
Report diarrhea, your immunosuppression drug levels in your blood can
drop
Diabetes People have developed increased blood sugars at high levels
of this medication and therefore we must watch the levels carefully
Blood sugars and drug levels are monitored with your regular blood
work
Muscle pain People have experienced sore muscles, weakness, and
tingling
The symptoms are usually associated with the high doses
Report your symptoms to the transplant team
Unusual effects Report symptoms that are of concern; you are on many
medications and your transplant team may be able to help alleviate
your discomfort
Sirolimus (Rapamycin) Sirolimus is a new medication, which is used to
prevent rejection, that is still being studied. It has been reported
to have anti-tumor activity. It decreases the ability of your body to
identify invading cells.
How to take
Must be taken daily at 8 a.m. during the initial post-transplant
phase; may change by 30 minutes per day to fit personal routines once
follow up is decreased.
Take consistently with or without food. Some patients need food to
avoid nausea.
Drug can be obtained only at the University of Alberta Hospital
Pharmacy.
Always make sure you have an adequate supply; out-of-town patients
can have supplies shipped if necessary.
On blood work days, always take Sirolimus after blood is drawn; you
will need to bring the medication to the lab with you.
Side Effects Gastrointestinal Diarrhea, nausea and vomiting can be a
problem
Take your medications with food if stomach upset is a problem
Report diarrhea as it may alter the immunosuppression drug levels in
your blood
High Lipid Level Sirolimus may raise your cholesterol and
triglycerides
Your lipids will be monitored on a monthly basis
Follow a low-fat diet to help reduce your levels
If required, medications will be used to help control elevated lipid
level.
Low Blood Cells Low white blood cell counts can occur due to the
suppression of the bone marrow which produces these cells.
Your blood counts will be monitored
If required, we will reduce your dosage to allow your blood counts to
rise
Skin Changes Soft fingernails have been reported
Keep nails trimmed short to minimize injury to the nail beds
Mouth Sores Some people have developed mouth sores
Report any sores in your mouth to the transplant team. If they occur,
stop using irritants such as regular toothpaste and use baking soda
or toothpaste with no sodium lauryl sulfate. If they persist we will
order some mouthwashes that will help you get rid of them.
Daclizumab (Zenapax) Zenapax is a genetically engineered antibody
that inactivates the building blocks of the immune system. This
protection for your islet cells lasts approximately four months,
decreasing the risk of acute rejection at the most vulnerable time
period following the transplant procedure.
How to take
Zenapax is given in 50cc of normal saline over fifteen minutes every
two weeks, for a total of five doses. This is administered in the
Medical Outpatient Unit. Vital signs are taken pre-infusion, post-
infusion, and fifteen minutes post-infusion.
Side Effects
There are no known side effects directly linked to Zenapax, but there
may be side effects due to the administration of a foreign protein,
including itching, rash, fevers, shortness of breath, back or flank
pain, or low blood pressure.
To date three serious side effects have occurred with patients taking
Zenapax - shortness of breath, low oxygen levels in the blood, and
renal vein thrombosis (clotting) - however, these could also be due
to many other underlying factors and therefore are not proven to be
caused by Zenapax itself.
Infection
Infection is a common complication after transplantation. As you have
already learned, your suppressed immune system has less ability to
fight the organisms in your body that cause infection. Your immune
system attacks any organisms that are identified as not belonging in
the body. When the immune system is unable to control an organism,
infection occurs. We use medications to prevent infection
(prophylaxis) and to treat infections (therapy).
You will know when you have an infection. When the body is fighting
an infection, there is cell damage and there are chemicals released
that cause symptoms. These symptoms are signs that your immune system
is fighting an organism, so it is important to know what to look for.
Signs of Infection
Increased temperature (higher than 38.5C), chills, and/or shivering
Pain, swelling, redness, and heat in any area of the body
Feeling very tired, achy, or weak
Vomiting or diarrhea
Frequent, painful urination or change in color, amount or odour of
your urine
Cold symptoms, green sputum (phlegm)
Sores on your lips, mouth, or face
Organisms are everywhere. On your skin, on surfaces you touch, in the
air you breathe, and in the food you eat. They also live in our
bodies. However, only a small number of organisms cause illness.
Organisms can enter your body from an injury to your skin or through
body cavities such as your nose or mouth which are exposed to the
environment. That is why good hygiene is an important way to prevent
infection. Handwashing is the most effective way of reducing your
risk of infection. Avoiding contact with those who are ill is another
common-sense way to stay healthy. Ensuring your body is healthy by
eating a good diet, getting adequate rest, exercising regularly, and
having regular dental care also help prevent infection.
Even with our best efforts to stay healthy, we still get infections.
Sometimes our immune systems need help in keeping infection under
control. The following is a description of the types of organisms
that cause infection. You will also learn about the most common
infections that occur after a transplant. Finally, we will tell you
about the drugs that you might need to manage infection.
Organisms that Cause Infection
Bacteria
Most bacteria are harmless or natural helpers to our bodies. We have
bacteria that live in and on our bodies that we need to stay healthy -
our natural "bacterial flora". For example, bacteria that live in
your gut help digest your food. Natural bacteria normally use the
resources in our body that harmful bacteria need to become
established and cause infection. Bacterial infections occur when
harmful bacteria do become established. We also get infections in our
bodies when our natural bacteria move to places they should not be.
For example, your natural skin bacteria are healthy on the outside of
your body but can make you very ill if they become established
inside. Bacterial infections are treated with antibiotics. There are
many different kinds that are effective against only some bacteria.
We will select the best antibiotic for the type of bacterial
infection that you have.
Viruses
Viruses are organisms that cause the common cold, influenza (flu) and
many childhood illnesses. Most viral infections last only a short
time. However, some viruses remain in our bodies forever. For
example, the virus that gave you chicken pox as a child remains in
the nerve roots of the spine and can cause shingles in adulthood.
Herpes simplex, the cold sore virus, resides in the nerves of the
face, causing flare-ups of new sores. These "resident viruses" can be
problematic after a transplant, especially in the early months when
the immunosuppression levels are high. The following provides more
detailed information about specific viruses we are concerned about.
Varicella Zoster
This virus causes chicken pox during the initial infection and
shingles later in life. If you have not had chicken pox, it is
important to notify the transplant team as soon as you know that
someone you have been around in the previous two weeks has developed
the infection. We will arrange for a shot of antibodies against the
virus (VZIG). The antibodies last about six months. After your
transplant, do not get vaccinated against chicken pox. The vaccine
has live, but very weakened chicken pox viruses. Since you are
immunosuppressed, even the weakened chicken pox viruses can cause
infection. If you develop shingles, we will treat the illness.
Shingles usually starts with a burning pain in your back, followed by
a breakout of very painful, fluid-filled blisters. Notify the
transplant team immediately of symptoms. Prompt treatment is
important.
Herpes Simplex
This virus causes painful sores on the mouth and genitalia. They can
spread to other parts of the body if left untreated. The sores
usually start with a burning, itchy feeling, development of redness
and swelling, followed by a fluid-filled vesicle, then a painful
sore. The sores usually take about two to three weeks to heal. Notify
the transplant team as soon as you develop the sores so that we can
start treatment.
Cytomegalovirus (CMV)
This is a very common virus that causes severe flu-like symptoms.
Most people eventually contract the virus, but are unaware that
the "flu" was a CMV infection. This virus flares up again in people
who are immunosuppressed. Your donor organ can also infect you. The
virus may cause muscle pain, weakness, tiredness, respiratory
symptoms, diarrhea, nausea, vomiting, hepatitis and a variety of
other symptoms. We monitor your blood work and provide preventative
medications if your donor was positive for CMV.
Epstein-Barr Virus (EBV)
EBV is commonly referred to as "mono" or "the kissing disease". Like
CMV, this virus is contracted eventually by most people. It causes a
flu-like illness and most people are not aware they have had it. Some
individuals become very ill (infectious mononucleosis) with severe
fatigue, sore throat, headache, hepatitis, possibly a rash, painful
muscles, fever, and achiness. You can become infected by your donor
organ if you have not already had the virus. If you are negative for
EBV, but your donor was positive, we monitor your blood work for the
virus and use preventative medications.
Fungus
Fungi also cause infections in our body. "Athlete's foot" is an
example of a fungus that causes infection. Yeast infection in the
mouth caused by an overgrowth of this naturally occurring fungus
sometimes occurs after a transplant. It is commonly called "thrush".
Symptoms include a sore mouth and white spots on the insides of your
cheeks and on your tongue. You should examine your mouth daily before
you brush your teeth. Once home, if you develop symptoms, we will
treat the infection. In addition, women will be at an increased risk
of vaginal yeast infections and are asked to alert the doctors if
this becomes a problem. Do not treat yeast infections yourself with
over-the-counter medications due to the negative interaction with the
immunosuppressant medications.
Parasites We have parasites that live in our lungs called
pneumocystis carinii. Given the opportunity, these parasites may
cause pneumonia (PCP). For the first six months after your transplant
when immunosuppression is highest, we will give you medication to
prevent the development of pneumonia from this organism.
You may be worried about these organisms and the risk of infection.
Remember that common sense is the best approach to caring for
yourself. Report any symptoms of infection to the transplant team,
follow the infection control suggestions, and relax. Even though you
are immunosuppressed, your body can still fight and control
infection, especially with a little help from you and your transplant
team.
Medications for Preventing and Treating Infection
Co-trimoxazole (Septra, Bactrim) Co-trimoxazole, is part of the
family of medications commonly called "sulfa drugs". It is used to
prevent pneumonia from pneumocystis carinii. You will take the
medication for six months after your transplant.
How to take
Taken once daily at the time you prefer
Side effects
Gastrointestinal People have reported stomach upset
Take with food if experiencing nausea
Skin Reaction Rash and itchiness may develop
Notify the transplant team immediately and we will use another
medication
Sun sensitivity is common
Avoid sun exposure, wear protective clothing and use SPF 15 sunscreen
Low Blood Cell Low white blood cell counts can occur due to the
suppression of the bone marrow which produces these cells, especially
with the effects of other transplant medications
We will monitor your blood counts and change medications if necessary
Renal effects This drug can stress the kidneys, especially in
combination with tacrolimus
Drink three litres of water daily to help protect your kidneys
Water is best; juices increase sugars and calories; coffee and tea
make you urinate
Try lemon or lime in water; keeping a sports bottle of water at your
side all day long helps increase water intake
We will monitor your kidney function and change medications if needed
Toxic Reaction In very rare instances, this drug can cause severe
toxicity with anaphylaxis (severe allergic reaction with difficulty
breathing) or significant symptoms of skin rash and illness
SEEK MEDICAL HELP IMMEDIATELY
Pentamidine
This medication is used to treat parasitic infections. It is used as
an alternative when co-trimoxazole is not suitable.
How to take Pentamidine is given by inhalation once a month for six
months after your transplant. You will go to the Respiratory
Department where a Respiratory Technician will administer the drug.
It is important to keep your appointments for the medication
inhalation, as long breaks between doses place you at risk for
pneumonia.
Side Effects Respiratory People find they cough during the inhalation
Slow, easy breaths help minimize the irritation
Pancreatitis In very rare circumstances, people have developed
redness, swelling and pain in the pancreas
Symptoms include severe upper abdominal pain, fever, chills, shakes,
and a feeling of being unwell
Notify your transplant team immediately
Ganciclovir (Cytovene)
This medication is taken by mouth and is used to prevent CMV and EBV
infections from your donor organ. In the event you develop infection,
larger doses given intravenously will be used to control the
infection.
How to take
When taken orally, you will take three doses daily: morning,
afternoon, and evening. You will be on the medication for twelve
weeks. If you develop an infection, the oral medication will be
stopped and you will be placed on intravenous ganciclovir for
fourteen days. The medication is given twice per day, about twelve
hours apart.
Side Effects
Low Blood cell Low white blood cell counts can occur due to the
suppression of the bone marrow which produces these cells, especially
with the effects of other transplant medications and the virus
We will monitor your blood counts and reduce the dose of the
medication if necessary
Renal effects This drug can stress the kidneys, especially in
combination with Tacrolimus
Drink three litres of water daily to help protect your kidneys
Water is best; juices increase sugars and calories; coffee and tea
make you urinate
We will monitor your kidney function and reduce the dose of the
medication if necessary
If you require IV Ganciclovir Phlebitis is redness, swelling, heat,
pain and hardness in a vein caused by the intravenous needle and the
medication
This is a common problem that requires multiple changes of the
intravenous site
To avoid this problem, you will have a PICC line placed - it is a
long catheter that is inserted through a vein in your arm and
threaded up to a large vein in your chest under the guidance of x-
rays
The PICC line will remain in place until your Ganciclovir therapy is
completed
Nystatin (Nilstat)
Nystatin helps prevent yeast infection in your mouth. If you develop
a thrush infection, you will be started on this medication.
How to take
The medication must be swished throughout your mouth, then swallowed
for effective prevention or treatment of infection. It is taken after
each meal, and before bed. Do not eat or drink anything for 30
minutes after your dose.
Side Effects
Gastrointestinal Nausea, vomiting and diarrhea sometimes occurs with
high doses
What about vitamins?
Until you are insulin free for three months, you will be on high
doses of vitamins. When the islet cells are transplanted, they are
injured and produce oxygen-free radicals that hurt the cells. There
is some evidence the vitamins may help reduce the level of these
toxic radicals. The vitamins have virtually no side effects taken in
the doses we prescribe.
Your Post-Transplant Schedule
Blood Testing
Every Monday, Wednesday and Friday you will have blood drawn in the
Clinical Investigation Unit (CIU) on 5E2 before 8:00 am. For these
tests you must be fasting and do not administer your morning insulin
until after the tests have been completed. In addition, do not take
your Tacrolimus or Sirolimus until after your blood work is done.
The frequency of blood testing will decrease over time as your islet
cells begin to work and your drug levels are stabilized. Eventually,
you can have your bloodwork done in a lab closer to your home.
For the first blood work of every month, you will have your lipids
tested. You will not be able to eat or drink anything other than
water for twelve hours before the test. No alcohol for thirty-six
hours before the blood test.
Keep track of your daily blood sugars and bring your records to the
appointments.
Clinic Appointments
You will meet Dr. Ryan and Dr. Shapiro at the Transplant Clinic every
week. They will tell you what changes to make to your medication and
how often you have to come for blood work and appointments.
Medication
Always keep a two-week supply of your medication on hand. Tacrolimus
and Sirolimus can only be obtained at the University Hospital
Outpatient Pharmacy. If you live outside of Edmonton you can make
arrangements for the pharmacy to ship the medication to you by
courier.
You are on Zenapax every two weeks for a total of five doses. The
first dose was at the time of transplant. Zenapax is given by
intravenous infusion on the Medical Outpatient Ward 4F2. Before you
go to the ward, please go to Admitting for admission and to receive
your paperwork. Your first appointment is two weeks following your
transplant. You will book the remaining appointments with the staff
in the Medical Outpatient Unit.
Your immunosuppressive medication places you at risk for developing
pneumonia from a parasite called pneumocystis carinni. You will
receive an inhaled antibiotic called Pentamidine on a monthly basis
for the first six months after your transplant. Please go to 3A4.08.
Your first appointment is within the first week following your
transplant. You will book the remaining appointments with the
respiratory therapist.
Glucose Monitoring
Initially, you must monitor your blood glucose levels seven times
daily. The frequency will be decreased over time as your glucose
levels stabilize. On the first two days of each month, you must
monitor before and after meals and before bed. We will obtain the
values when your glucometer is downloaded.
Rejection
We will know you are rejecting your new islet cells when they no
longer produce insulin. By the time this happens it will probably be
too late to treat the rejection medically. It is therefore very
important to prevent rejection by maintaining adequate
immunosuppression and preventing infections.
Infection
The drugs you are on to stop rejection from happening suppress your
immune system. This means that you must take care to prevent or get
early treatment for any infections you may be experiencing. If you
have any symptoms of infection- fever, vomiting, diarrhea or pain-
please see your doctor immediately and notify your transplant team.
Additional Tests
There are a variety of other tests that will be completed throughout
the next five years after your transplant. These tests will be booked
at intervals that are consistent with your first transplant and the
date you become insulin free. We will work with you to schedule the
appointments as conveniently as we can. Here is an example of a
schedule:
Follow-up Studies and Time Required
Test Name
Purpose
Substance Injected or Ingested
Blood removal per sample
Frequency and time per test
Capillary blood glucose on finger stick samples via glucometer
To monitor /assess
Blood glucose levels
N/A
0.1 to 0.5 ml
7x/day until off insulin. 4x/day for the first year then once a day
for 5 years. Doing 7 test for 2 days the first of every month for the
entire study.
Regular blood work: CBC, glucose, C-peptide, creatinine,
electrolytes, Tacrolimus and Rapamycin levels
To assess insulin output, renal function and drug levels
N/A
7 to10 ml
3x/week for 2 weeks. 2x/wk for 1 month then weekly until three
months then once every 2 weeks till 6 months and then monthly
Fasting triglycerides and cholesterol, HbA1c
Assess impact on blood lipids and adequacy of glycemic control
N/A
3 to 5 ml
Monthly for 5 years.
Meal replacement challenge
To assess insulin output/ glucose
360 ml of Ensure
3 ml x 2
90 minutes. Between transplants, at 2 weeks, one month and months
3,6,12 and then every 6 months thereafter.
Oral glucose tolerance test (OGTT)
To assess insulin secretion/ glucose control after oral glucose
1gm/kg of dextrose by mouth
4 ml x 5
2.5 hours. Every 6 months for 5 years
Arginine and Intravenous glucose tolerance test
To assess insulin secretion/ glucose control after intravenous
arginine and glucose
Arginine 5g Glucose 0.3g/kg of dextrose intravenously
4 ml x 23
3 hours. Between transplants, 1,3,6,12 months and then every 6
months for 5 years
Retinal Fundus examination
To check stability of retinal blood vessels
N/A
N/A
Every three months for first year and then yearly
Nerve conduction studies
To assess effects on nerve function
N/A
N/A
Once a year for 5 years
Physical examination
To assess impact of the treatment for control of diabetes
complications
N/A
N/A
Once a year for 5 years
Clinic interview and examination
To review effects of the therapy and glucose control
N/A
N/A
1x/week for first month. Every 2 weeks until 3 months and then
monthly.

Life after the Islet Cell Transplant

2007-11-25 23:19:57

Ruthie,
I am going to try to give you what you requested.
Thanks,
Robert
A Founding Member of TPP,
roberthammett@...

Re: [ThePancreatitisPlace] HELPING OTHERS !

2007-11-25 19:16:55

Thanks for your message. I have pancreatitis caused by
Medication. I try to minimize my medication use to a
minimum amount daily. I have not written down
everything that I have eaten, but I have researched as
much as possible. Another good resource is
www.medifocus.com "Chronic Pancreatitis"
Thanks
David

Keeping Diabetes at Bay After Transplant

2007-11-25 10:25:01

http://www.wmtw.com/global/story.asp?s=1610500&ClientType=Printable
Keeping Diabetes at Bay After Transplant
WEDNESDAY, Jan. 21 (HealthDayNews) -- In research with mice, a new
anti-inflammatory drug called lisofylline prevented diabetes
recurrence after transplantation of insulin-manufacturing pancreatic
islet cells.
University of Virginia Health System researchers report their finding
in the Jan. 20 issue of Transplantation.
In recent years, pancreatic islet cell transplantation has become a
promising treatment for people with type 1 diabetes. However, without
powerful immunosuppressive drugs, those transplanted islet cells
would be destroyed by the body's immune system.
This study suggests lisofylline may have the potential to help
prevent this destruction by preserving insulin secretion by
pancreatic beta cells in the presence of autoimmune attackers called
inflammatory cytokines.
"Our findings are very encouraging and we are excited that
lisofylline worked so well in this animal model," Dr. Jerry Nadler,
chief of the University of Virginia's division of endocrinology and
metabolism and director of the Diabetes and Hormone Center of
Excellence, says in a prepared statement.
"We have discovered a potentially new way to protect islet cells in a
clinical transplant setting. It's possible this research could form a
basis for additional studies to use [the drug] or related anti-
inflammatory compounds in humans to limit the need for more toxic
immunosuppressant drugs in islet cell transplant patients," Nadler
says.

Islet Cell Transplant

2007-11-25 00:23:02

http://www.organtx.org/tx/isletcell.htm
Transplant Breakthrough May Lead to Potential Diabetes Cure!
Satellite Feed 5-13-03 Subject: Interventional Radiology Technique
Provides Safe Reliable Transplantation of Insulin Producing Islet
Cells Into the Liver Without Surgery; First of Three Cutting Edge
Medical Feeds in May NEW YORK, /PRNewswire/ -Source: Society of
Interventional Radiology- A new clinical trial shows unstable Type-1
diabetics can stop regular insulin shots and become insulin free
after islet cells are transplanted into their liver.
The Society of Interventional Radiology reports that one year after
undergoing an islet cell transplant, 81 percent of the patients are
insulin free. This is a major medical advancement that could lead to
helping the 1 million Americans who suffer from Type-1 diabetes, the
condition in which the body produces no insulin which is necessary
for the body to use sugar-the basic fuel for the body's cells.
Interventional radiology allows the cells to be placed directly in
the liver without surgery, where they can begin producing insulin
almost immediately. The study shows that the interventional radiology
delivery technique provides reliable and safe access to the liver,
and together with islet cell separation techniques and
immunosuppressive treatment, offers a significant development in the
treatment of Type-1 diabetes.
During the two-hour minimally invasive procedure patients are awake
and can watch the procedure on a monitor. Because of this non-
surgical technique, recovery is rapid and patients can go home the
next day. The feed includes bites with transplant team doctors and a
Type-1 diabetic of 23 years who's been insulin free for one year
after two islet transplants, as well as b-roll footage of the
hospital, the patient and the interventional radiology suite. A list
of transplant centers for patients is available at www.SIRweb.org on
the homepage under "Hot Topics."
Two additional cutting edge medical stories focus on: Minimally
invasive laser treatment of varicose veins which affect half of all
people 50 and older. Fertility results after non-surgical treatment
of uterine fibroids (UFE) that affect up to 40 percent of all women
35 and older and result in 200,000 hysterectomies each year. These
topics feed on May 20th (1:30-2:00 PM ET) and 21st (10:30 - 11:00 AM
ET) with the same satellite coordinates as the above mentioned
project.
These feeds are provided by the Society of Interventional Radiology
(SIR) for your free and unrestricted use. Interventional Radiologists
are board certified doctors who specialize in minimally invasive,
targeted treatments performed using imaging guidance. They use their
expertise in reading x-rays, ultrasound, MRI, and other diagnostic
imaging, to guide tiny instruments such as catheters, through blood
vessels or the skin to treat diseases without surgery.
Promising Results for Islet Cell Transplantation to Treat Diabetes
May Lead to Potential Cure - Interventional Radiology Technique
Provides Safe and Reliable Delivery of Cells to the Liver Without
Surgery - SALT LAKE CITY 3-28-03 /PRNewswire/ -- Early data shows
that transplanting healthy, insulin producing islet cells by infusion
into the portal vein to the liver enables uncontrolled type 1
diabetic patients to become insulin free, according to data presented
here today at the 28th Annual Scientific Meeting of the Society of
Interventional Radiology. "This work is the collaborative effort of
transplant specialists and interventional radiologists. This early
data shows that if we can get enough healthy islet cells to the
liver, diabetes patients could potentially be cured," says Richard
Owen, M.D., interventional radiologist at the Alberta Hospital. The
islet cells become rapidly engrafted into the liver and secrete
insulin almost immediately. The study shows that the interventional
radiology delivery technique provides reliable and safe access to the
liver, and together with islet cell separation techniques and
immunosuppressive treatment, offers a significant development in the
treatment of type 1 diabetes.
Interventional radiologists specialize in performing minimally-
invasive treatments using guided imaging to treat diseases without
surgery. Using ultrasound or fluoroscopic guidance, a needle is
placed through the skin to a branch of the right portal vein, where
the cells are infused. "Interventional radiology allows the cells to
be placed directly where they need to engraft to begin producing
insulin for the patient. This minimally invasive technique is a safe,
accurate and practical means of delivery in the hands of a skilled
interventional radiologist," says Dr. Owen, in his scientific
presentation New Frontiers in Interventional Radiology. The portal
vein is the site of islet implantation because of the ease of
technical access combined with cumulative data indicating that this
route is the safest and most durable route for transplantation. The
procedure is well within the capability of all interventional
radiologists, and with adequate cell separation and purification, the
procedure and clinical results of this study should be reproducible.
About The Study and Islet Cell Transplantation: Forty-eight patients
had 90 transhepatic portal vein islet cell transplantation procedures
for type 1 diabetes. Twenty-two patients had two transplants, 10
patients had three transplants, and sixteen patients had a single
transplant. In most cases, more than one transplant is required to
obtain a sufficient number of islets to become insulin independent.
On average, only about 5,000 islet equivalents per kilogram can be
obtained from one donor pancreas. Successful islet cell infusion was
achieved in all cases by the interventional radiologist. All of the
patients who received more than 9000 islet equivalents per kilogram
became insulin independent. One year later, twenty-one of the twenty-
six patients that have received the full number of islets (81
percent) are insulin free. Most importantly, they are now
metabolically stable.
"The people who took part in this trial had labile diabetes, which is
dangerously unstable metabolic control. They could no longer tell
when they had dangerous lows in blood sugar and had to test
continuously. These patients were losing consciousness off and on,
were at risk for diabetic coma, and were afraid to go to sleep at
night, for fear that they would not wake up. Islet transplantation
has given them their lives back," says Owen.
Treatment Availability: This is a very active area of research and
the Alberta hospital in Edmonton, Canada is one of several hospitals
in the United States and Canada with islet cell transplantation
programs. Although not yet available outside of a clinical trial,
multi-national trials using the Edmonton protocol are now in
progress. This treatment is still being studied and is reserved for
diabetics who have unstable diabetes to such an extent that the risks
of transplantation are considered less than the risk of uncontrolled
diabetes.
Recent developments in tailored immunosuppression for islet
transplant recipients, combined with the delivery of sufficient
islets, has led to rejuvenated enthusiasm for clinical islet
transplantation as a potential therapeutic option for selected
patients with type 1 diabetes. This enthusiasm is based on current
insulin independence rates at one year exceeding 80 percent, combined
with the minimal morbidity of the interventional approach for
implantation. Improved safety, avoidance of major surgery, rapid
hospital discharge and return to normal activity has been a major
advantage of this approach, making the procedure attractive to
patients.
These new success rates are very encouraging. Prior to these recent
developments, the previous success rates were about 7 to 14 percent.
There are still limitations to the procedure. Isolating the cells
from a donor remains a highly technical and labor intensive process.
If a large enough number of islets can be isolated then they are
transplanted. This early work shows that if we can get enough healthy
islet cells to the liver, diabetic patients could potentially be
cured of their need for insulin.
About the Society of Interventional Radiology: An estimated 5,000
people are attending the Society of Interventional Radiology 28th
Annual Scientific Meeting in Salt Lake City. The Society represents
interventional radiologists -- physicians who specialize in minimally
invasive, targeted treatments performed using guided imaging.
Interventional radiology procedures are a major advance in medicine
that do not require large incisions -- only a nick in the skin about
the size of a pencil tip -- and offer less risk, less pain and
shorter recovery times compared to surgery. Interventional
radiologists pioneered modern medicine with the invention of
angioplasty, the first catheter-delivered stent and the coronary
angiography technique most used worldwide -- state of the art
treatments that are commonplace in medicine today. More information
can be found at www.SIRweb.org SOURCE Society of Interventional
Radiology
Emory performs Georgia's first islet cell transplant 3-25-03 The
Telegraph, Macon - Emory University has performed the state's first
islet cell transplant procedure, which can help end diabetics'
dependence on insulin shots to control blood sugar.
Wendy Kenny, a 42-year-old Covington pharmacist, received the
procedure Thursday. Kenny was diagnosed with type 1 diabetes at age
9. Type 1 diabetes usually occurs early in life. People with the
condition are unable to manufacture their own insulin because their
pancreatic islets don't function.
In the procedure, islets that can restore normal insulin production
are harvested from a donor pancreas and injected through a small
incision in the liver of the recipient. Emory officials hope to do
three or four transplants under the clinical trial that included
Kenny's procedure and possibly another 10 transplants in the future.
Pancreatic Islet Transplantation Produced Insulin Independence In
Type I Diabetics -- A DGReview of :"Achievement of insulin
independence in three consecutive type-1 diabetic patients via
pancreatic islet transplantation using islets isolated at a remote
islet isolation center." Transplantation 1/10/03 by Mark Greener -
Early data demonstrate that pancreatic islet transplantation produces
insulin independence in patients with type-1 diabetes.
Researchers from Baylor College of Medicine, Houston, Texas, United
States, performed pancreatic islet transplantation in three patients
with type 1 diabetes. They also had histories of severe hypoglycaemia
and metabolic instability. Islet isolation, at a site distant from
the transplantation centre, began within 6.5 hours.
All three patients attained sustained insulin independence after
transplantation of between 395,567 and 563,206 pancreatic islet
equivalents (IEQ). Two patients underwent a second islet transplant
with 326,720 and 768,132 IEQ to increase functional pancreatic islet
reserve. Follow-up was between 0.5 and 4 months.
Mean glycosylated haemoglobin values and the mean amplitude of
glycaemic excursions declined in two and three patients respectively.
Islet allografts responded to in vitro glucose stimulus before and
after shipment to the transplant centre. No patient experienced
hyperglycaemic or hypoglycaemic episodes since the transplant. No
complications occurred.
The authors came to three conclusions: first, pancreatic islets
remain viable after shipment to remote sites for transplantation;
second, a small number of regional units could supply isolated
pancreatic islets to remote transplant centres and third, that
pancreatic islet transplantation performed by remote centres can
produce insulin independence.
Transplantation 2002;27:1761-6. "Achievement of insulin independence
in three consecutive type-1 diabetic patients via pancreatic islet
transplantation using islets isolated at a remote islet isolation
center."
15 Million Clinical Center for Islet Transplantation Launched by the
Juvenile Diabetes Research Foundation at University of Alberta - New
Center Expands Promising Edmonton Protocol Research - NEW YORK 11-8-
01 /PRNewswire/ -- The Juvenile Diabetes Research Foundation
International (JDRF) announced today a $15 million grant to open a
clinical center at the University of Alberta, designed to study the
treatment of juvenile diabetes (also known as Type 1 diabetes)
through islet transplantation. The five-year grant is intended to
sustain research into furthering the historic advances of the
Edmonton Protocol diabetes research. The grant was announced at a
JDRF fundraising gala this evening in Toronto, Canada.
The Edmonton Protocol identified islet transplantation as a promising
area of research. It established a set of procedures for islet
transplantation from donor pancreases that can restore normal insulin
production in people with Type 1 diabetes. There are still several
problems associated with islet transplantation and the Center aims to
conduct various research initiatives to address these issues.
"The Edmonton Protocol was a major breakthrough in the path to
finding a cure for diabetes-a disease which affects more than 16
million Americans and 120 million people worldwide. We hope that the
development of this new clinical center will enable JDRF-funded
researchers to make even further strides in understanding how islets
can be used to cure Type 1 diabetes," said Charles J. Queenan III,
Chair of Research for JDRF. "JDRF has long been at the forefront of
funding cutting-edge diabetes research and our goal is to quickly
move more research results into clinical applications."
Overcoming limitations of the Edmonton Protocol - The challenges
associated with the Edmonton Protocol will be paramount to the
research conducted at the Center. These include the fact that two
donor pancreases are required to transplant one patient and there are
relatively few pancreases available. Secondly, it is still difficult
to extract islets from the donated pancreas in a way that makes them
pure enough for transplantation.
Thirdly, researchers cannot distinguish well between high quality
islets, which are very likely to survive transplantation, and low
quality islets, which are not. Finally, researchers need to find a
way to overcome "allograft rejection," the body's normal defenses
that turn on against foreign tissues whenever any kind of transplant
takes place. The Center will accomplish this by testing safer and
more effective immunosuppressive drugs.
"The Edmonton Protocol helped us to understand how we can use islets
to treat people with Type 1 diabetes," says James Shapiro, MD, PhD,
Director of JDRF Clinical Center, University of Alberta. "Now we need
to concentrate on how to alleviate the problems still associated with
this procedure. We are confident that with the support of this new
Center, we will be able to put an increased emphasis on identifying
solutions to these issues."
Clinical Center focus areas - The Juvenile Diabetes Research
Foundation Clinical Center research will begin work immediately. The
new projects that will be undertaken at the Center will include three
focus areas. The first will focus on improving the safety, quality,
and effectiveness of transplanting islets from donor pancreases. The
second is on islet transplants and the complications associated with
diabetes. This study area will examine Edmonton Protocol patients to
determine whether or not the restoration of normal insulin also
reverses the complications associated with diabetes. The third focus
area will look at the quality of life and cost of islet
transplantation. Researchers will investigate how patients feel
before and after transplant and they will also examine economic
evaluations of the transplant procedure.
"These studies are central to further understanding how we can use
islet transplantation to cure people with Type 1 diabetes, and will
help to determine the plausibility and economic realities of the
procedure," says Dr. Shapiro.
Established in 1908, the University of Alberta has grown to be one of
Canada's largest research-intensive universities, with external
research funding in 1999/2000 or more than $213 million (CD). The
University is located in Edmonton, the capital of the province of
Alberta. The University of Alberta serves more than 30,000 students
in 200 undergraduate programs and 170 graduate programs.
JDRF, the world's leading nonprofit, nongovernmental funder of
diabetes research, was founded in 1970 by the parents of children
with juvenile diabetes-a disease which strikes children suddenly,
makes them insulin dependent for life, and carries the constant
threat of devastating complications. Since inception, JDRF has
provided more than $500 million to diabetes research worldwide. In
2001 alone, the Foundation provided $115 million and 87 cents of
every dollar goes directly to research and education about research.
JDRF's mission is constant: to find a cure for diabetes and its
complications through the support of research. For more information,
visit the website at http://www.jdrf.org, or call 800-533-CURE.
SOURCE Juvenile Diabetes Research Foundation Int
l Incara's Catalytic Antioxidant Preserves Human Islets Isolated from
Organ Donors - Data presented to the Cell Transplant Society 10th
Anniversary Meeting RESEARCH TRIANGLE PARK, N.C., Oct. 16
01 /PRNewswire/ -- Incara Pharmaceuticals Corporation (Nasdaq: INCR)
announced that its prototype catalytic antioxidant AEOL 10113
produced an approximate 3-fold increase in the survival of human
pancreatic islets when maintained in culture for up to 6 days with no
loss of beta cell function. Islets are anatomical structures in the
pancreas that contain beta cells, which make and secrete insulin to
regulate blood sugar levels. These data were presented at the Cell
Transplant Society 10th Anniversary Meeting in Keystone, Colorado on
October 16, 2001 by Jon Piganelli, Ph.D. and others from The Diabetes
Institute, University of Pittsburgh, Pittsburgh PA.
"Islet transplantation, a potentially curative treatment for Type 1
diabetes, is limited by the ability to isolate functional human
islets from donors. Currently, islets from two to four organ donors
are needed to perform a transplant into one patient," stated Richard
E. Gammans, Ph.D., MSM, Senior Vice President of Antioxidant
Therapies at Incara. "An agent that can significantly increase the
number of functional human islets available for transplantation would
represent an important advance in islet transplant treatment. These
data also suggest other applications of our catalytic antioxidant
compounds in the management of diabetes and in tissue and organ
transplant therapies."
Type 1 or juvenile diabetes is an autoimmune disease in which the
pancreatic beta cells, which produce and release insulin, are
destroyed. Type 1 diabetics are dependent on daily insulin injections
to stay alive. Onset usually occurs during adolescence or young
adulthood. Diabetic complications int's blood sugar without the need
for daily insulin injections.
"These data suggest a role for our catalytic antioxidants in cell and
organ transplant. In addition to pursuing the effect on human islets,
we are investigating the use of our catalytic antioxidants in our
liver stem cell program," stated Clayton I. Duncan, President and CEO
of Incara. "Our strategy is to leverage our technology platform to
address prevalent metabolic diseases such as diabetes."
Incara Pharmaceuticals Corporation (www.incara.com) is developing
therapies focused on tissue protection, repair and regeneration. This
includes developing a series of catalytic antioxidants for protection
of cells from damage such as that occurring in stroke and cancer
radiation therapy, and also protection of cells from transplant
rejection. Incara is developing liver progenitor cell therapy,
sometimes referred to as adult liver stem cell therapy, for treatment
of liver failure. In addition, Incara is conducting a Phase 2/3
multicenter clinical trial for OP2000, an ultra-low molecular weight
heparin being developed with Elan Corporation for treatment of
ulcerative colitis. Additional background information regarding
Incara's programs is available on the company's website at
www.incara.com.
The statements in this press release that are not purely statements
of historical fact are forward-looking statements, and actual results
might differ materially from those anticipated. These statements and
other statements made elsewhere by the company or its
representatives, which are identified or qualified by words such
as "intends," "likely," "will," "suggests," "expects," "might," "may,"
"believe," "could," "should," "would," "anticipates" or "plans," the
negative of those terms or similar expressions, are based on a number
of assumptions that are subject to risks and uncertainties. Important
factors that could cause results to differ include risks associated
with uncertainties of scientific research, clinical trials, product
development activities and the need to obtain funds for operations.
These and other important risks are described in Incara's reports on
Form 10- K, Form 10-Q and Form 8-K and its registration statements
filed with the Securities and Exchange Commission. Readers are
cautioned not to place undue reliance on these forward-looking
statements, which speak only as of the date hereof. The company
assumes no obligation to update the information in this release.
SOURCE Incara Pharmaceuticals Corporation
UT gets $1.7 million for cell transplant diabetes treatment 10-13-01
By Mary Powers powers@..., GoMemphis.com -- The
University of Tennessee Health Science Center received a $1.7 million
federal grant to serve as one of 10 national islet transplant centers
designed to make the experimental diabetes treatment more widely
available.
The money will go to expand laboratory space, staff and logistical
support so Memphis can serve as a regional supplier of the insulin-
producing islet cells. Researchers hope the donor cells will prove
effective at curing diabetes or significantly reducing a patient's
risk of blindness, kidney failure and other disease complications.
The grants were announced Friday as a new era of islet cell
transplantation is poised to begin. The Food and Drug Administration
has approved about 30 islet cell transplant programs nationwide.
Transplants are under way in about a half-dozen.
The UT Health Science Center is home to one of those programs. Dr.
Osama Gaber, UT transplant division chief, said the university has
approval to transplant up to 12 patients. They will likely begin next
year.
Because it costs about $1 million to establish a lab to prepare islet
cells for transplantation, Gaber said the federal grants are designed
to build networks to provide cells for transplantation. Researchers
in Memphis and three other UT campuses are involved in the effort.
The approach is appealing because, unlike treating a patient's
diabetes by replacing the entire pancreas, islet cell transplantation
is an outpatient procedure that requires less suppression of a
person's immune system.
Heme Oxygenase-1 Protects Islet Cells From Destruction Following
Transplant WESTPORT, CT (Reuters Health) Oct 01 01 - Induction of
heme oxygenase-1, a ubiquitous stress protein, protects islet cells
from local inflammation and improves the function of such cells after
transplant in rodents, according to data published in the September
issue of Diabetes.
The findings suggest that "strategies aimed at inducing heme
oxygenase-1 upregulation might result in improved success in islet
transplantation" as a treatment for diabetes in humans, conclude Dr.
Luca Inverardi, of the University of Miami School of Medicine in
Florida, and colleagues there and at Harvard Medical School in
Boston.
The researchers examined the role of heme oxygenase-1 upregulation in
the protection of transplanted islets both in vitro and in mice. They
note that prior studies of this molecule suggested that it has potent
anti-inflammatory properties and could be useful in this setting.
Indeed, heme oxygenase-1 upregulation caused a reduction in
inflammation- or Fas-associated apoptosis in vitro and an improvement
in islet cell function after transplantation in vivo.
"Islet transplants are susceptible not only to the occurrence of
rejection and recurrence of autoimmunity in type 1 diabetes patients,
but also to nonspecific inflammatory events that take place at the
site of implantation, which might contribute significantly to graft
failure," Dr. Inverardi and colleagues note. The ability to reduce
the vulnerability of these cells to local inflammation could have
important implications, they say, for the success of islet cell
transplantation.
Source: Diabetes 2001;50:1983-1991.
Islet transplants offer hope that diabetes can be cured - The
experimental treatment is hailed as a breakthrough. 6-22-01 By Marie
McCullough, INQUIRER STAFF WRITER - The Philadelphia Inquirer -
Robert Teskey began to dread going to sleep as the diabetes that had
defined his life became almost impossible to control, despite maximum
insulin doses. The lawyer from Edmonton, Canada, had to set an alarm
for the middle of the night, check his blood sugar and force himself
to eat something. If he didn't, he might slip into a diabetic coma
and never wake up. Now, two years later, Teskey, 55, is among a tiny
but growing group of Type 1 diabetes patients who appear to have been
cured by experimental transplants of islets, the pancreas cells that
produce insulin.
"After my third transplant, I've never taken another injection of
insulin," said Teskey, who had battled so-called juvenile diabetes
since he was 14. "I can eat whatever I want. And I don't have to eat
all the time." The long-sought breakthrough in treatment, announced
last year by a research team led by James Shapiro at the University
of Alberta in Edmonton, will be a hot topic at the American Diabetes
Association's annual meeting, which gets under way today in
Philadelphia.
Experts stress that transplants are suitable only for a minority of
people with Type 1 diabetes, which is the least common of the two
types of diabetes, afflicting less than 10 percent of all patients.
Still, patients and their families are thrilled by the promise the
new therapy holds. "There's still a lot to be learned, but this is
the most optimistic I've been in 30 years," declared Lee Ducat, the
Philadelphia-area mother who founded the Juvenile Diabetes Foundation
after her son's diagnosis. "To take patients who are terribly ill and
going in and out of shock and give them a normal life . . . this is
an unbelievable result. They say they never knew what feeling normal
is all about."
The "Edmonton protocol," initially tested on Teskey and seven others,
has now been used on 17 Canadian patients. Only two have rejected the
transplants, according to preliminary reports. At least 11 U.S.
patients have been successfully treated at the University of Miami
and a few other institutions. Now, 10 scientific teams - four in
Europe and six in the U.S. - are poised to test the protocol on 40
more patients, using funding from the National Institutes of Health
and the Juvenile Diabetes Foundation. The foundation is also funding
trials of slightly different protocols at the University of
Pennsylvania and three other centers. "This is a remarkable advance.
It is very exciting technology," said Penn surgeon Ali Nagi, who has
been studying islets for 30 years.
More than a million Americans and 600,000 Canadians have Type 1
diabetes, sometimes called juvenile diabetes although people of any
age can get it. For unknown reasons, their immune systems destroy
their islets, which secrete insulin, a hormone essential to
converting blood sugar into energy. (In Type 2 diabetes, which is
often linked to obesity, the body doesn't produce enough insulin or
use it adequately.)
Type 1 patients must inject insulin daily and regulate blood sugar by
monitoring diet, exercise and stress. Even so, many ultimately go
blind, develop kidney failure or lose limbs because of circulation
problems. Patients are usually aware of dangerous drops in blood
sugar because of symptoms such as sweating and disorientation. But
those like Teskey become insensitive - "brittle," doctors call it -
to this fluctuation.
Before Edmonton, attempts to fix this metabolic disorder with islet
transplants were disappointing. Still, the new protocol poses both
medical and technical obstacles. While the actual operation is minor -
basically, a transfusion - patients must take immune-suppressing
drugs indefinitely to prevent their bodies from rejecting or
destroying the islets. The drugs cause side-effects such as mouth
sores, and put patients at risk of infection and illness.
That's why transplants are currently considered only for "brittle"
patients and those who need a kidney transplant, which also requires
immune suppression. "We need to be able to do this [transplant]
without long-term immunosuppressants," said Hugh Auchincloss, who is
leading a transplant trial and heads the Juvenile Diabetes Foundation
Center for Islet Transplantation at Harvard Medical School. "There
are 57 ways to do it in mice, but none yet in humans. And to be
honest, we don't see a clear answer at this point."
Another problem: Islet cells are scarce (only 2 percent of all cells
in the pancreas) and difficult to obtain from donated cadaver
pancreases, the current source. Researchers are trying to develop
other sources such as genetically engineered liver cells, stem cells,
or pig pancreases. The Edmonton protocol "was a big step forward, but
we still have a long way to go to cure diabetes," Auchincloss said.
Pancreas Islet Allotransplantation Maintained With Two Weeks Of
Immunosuppression June 4, 2001 WESTPORT (Reuters Health) -
Researchers have for the first time succeeded in transplanting and
maintaining isolated pancreas islet cells into diabetic rhesus
monkeys without long-term sustained immunosuppressive therapy,
according to a report in the June issue of Diabetes.
Dr. Francis T. Thomas, of the University of Alabama at Birmingham,
and colleagues induced diabetes in 11 rhesus macaques with
intravenous streptozotocin. Islet cell donors were selected to have
multiple donor major histocompatibility complex mismatches with the
diabetic recipients.
For immunosuppression, "we used a new drug we developed which wipes
out the CD3-epsilon receptor of T cells, including most importantly
the T cells in the sessile compartments, such as the lymph nodes and
spleen," Dr. Thomas told Reuters Health. The research team also used
15-deoxyspergualin (DSG) after discovering that it blocks maturation
of dendritic cells that present foreign antigens to T cells, Dr.
Thomas said.
A 2-week tolerance induction protocol was initiated on the day of
transplantation. Three protocols were used, anti-CD3 or DSG, or both.
The macaques also received methylprednisolone on days 0 to 2. Within
3 days after the transplant, the recipients exhibited normal
nonfasting blood glucose levels in the absence of exogenous insulin,
the investigators report. All seven animals that received the
combination tolerance induction protocol maintained prolonged graft
survival, four for more than a year. The remaining four macaques
failed to become long-term survivors.
None of the recipients exhibited IgG- or IgM-positive flow cytometry
antidonor crossmatches, although long-term survivors were
immunocompetent with respect to a microbial antigen. Between 6 months
and a year after transplantation, peripheral and total T-cell counts
recovered to their pretransplant levels. T-cell amplification was
limited, as seen by a lower response to an antigen to which the donor
were previously unexposed, Dr. Thomas' group notes. The investigators
also observed "prominent and sustained expression" of interleukin-4
and -10, and normal levels of gamma-interferon. These latter findings
indicate downregulation of peripheral T-helper-2 responses, the
investigators write. This suggests, they add, that "an
immunoregulatory rather than a deletional process underlies this
operational tolerance model."
Dr. Thomas noted that the islet transplantation procedure does not
require hospitalization, and can be done in a radiology department
using duplex Doppler to image the portal vein, into which the cells
are injected. "The patient then gets off the table and goes home," he
added. Dr. Thomas's group has also demonstrated the potential for
using live donors, having achieved successful transplantation of two
animals from one donor pancreas. He said that they have applied to
the FDA to begin testing the new CD3 immunotoxin in a phase I
clinical trial. Diabetes 2001;50:1227-1236.
Single-Donor Islet Transplantation Provides Insulin Independence -
Transplant 2001 By W. A. Thomasson - Special to DG News - CHICAGO,
IL -- May 15, 2001 -- Researchers reported on the first three
patients with type 1 diabetes in whom an islet-cell graft from a
single donor produced prolonged reversal of their disease. A number
of other diabetic patients have achieved prolonged insulin
independence, but these patients, mostly at the University of
Edmonton, in Edmonton, Canada, required multiple grafts from two or
more donors to achieve this goal. Bernhard Hering, MD, and colleagues
at University of Minnesota in Minneapolis, Minnesota, and University
of California, San Francisco, California, presented their findings at
the Transplant 2001 meeting, in Chicago, Illinois.
In developing their transplantation protocol, Dr. Hering and
colleagues addressed optimization of several different areas-pancreas
preservation, islet processing and testing, timing of islet infusion
and induction immunotherapy. They selected what appeared to be the
optimum technique in each area, and it is not clear which was key to
success, or whether the key is a combination of all. To date,
transplants have been done in three patients with type 1 diabetes who
produced no insulin (no C-peptide) and failed to recognize their
hypoglycemic episodes. The number of cells transferred ranged from
8,200 to 15,200 islet equivalents/kg body weight.
All these patients are now insulin-independent at follow-up times
exceeding 240 days for the first two patients and 180 days for the
third. However, the first patient, who received the fewest cells,
produces less insulin than the other two and has impaired glucose
tolerance not requiring treatment. Hemoglobin A1c, a measure of
glycemic control, is within the normal range for all patients. One
patient developed a generalized rash, and all three patients
developed neutropenia. In one case the neutropenia clearly met the
definition of a serious adverse event and in another it was
borderline. In all cases, however, neutropenia responded quickly to
treatment. There have been no rejection complications. Dr. Hering
noted that the Edmonton protocol has been used successfully in a
large number of patients and is therefore appropriately the basis for
the current multi-center trial. Nevertheless, this paper provides
further evidence that we may be approaching the point where type 1
diabetes will be considered a curable disease, he added.
Islet Autotransplantation Combined With Total Pancreatectomy Shows
Benefits A DGReview of :"Pancreas Resection and Islet
Autotransplantation for End-Stage Chronic Pancreatitis" - Annals of
Surgery 4-5-01 By James Adams
Patients with end-stage chronic pancreatitis who undergo islet
autotransplantation combined with total pancreatectomy show decreased
daily insulin requirements and decreased glycosylated hemoglobin
levels. In most cases, however, patients who undergo the combined
procedure compared remain insulin-dependent, researchers report.
Results come from a study of 24 patients who underwent the combined
procedure conducted by investigators at Leicester General Hospital
and University of Leicester, in Leicester, United Kingdom. Patients
underwent the combined procedure during a 54-month period. Early
complications included duodenal ischemia, a wedge splenic infarct,
partial portal vein thrombosis and splenic thrombosis. Long-term
problems were most often the result of intra-abdominal adhesions.
Four patients developed chronic abdominal pain and remained opiate-
dependent. Eight patients developed short-term insulin independence,
and three developed insulin independence. Overall, insulin
requirements and glycosylated hemoglobin levels decreased
significantly in patients who underwent the combined procedure
compared with those who underwent total pancreatectomy alone. "A
prospective, randomized study is therefore needed to assess the long-
term benefit of total pancreatectomy and islet autotransplantation on
diabetic complications," the investigators conclude.
Ann Surg 2001; 233: 423-431. "Pancreas Resection and Islet
Autotransplantation for End-Stage Chronic Pancreatitis"
Islet Cell Transplantation Showing 'Long-term' Results WESTPORT
(Reuters Health) Apr 03 01 - A total of 15 patients with unstable
diabetes have undergone islet cell transplantation at the University
of Alberta in Edmonton in the past few years. All became insulin-
independent after the procedure and 12 have remained so some for
more than 2 years.
Dr. James Shapiro reported the results of his team's experience at
this year's Experimental Biology 2001 meeting in Orlando this week.
Dr. Shapiro said that islet cell transplantation initially resulted
in all patients becoming insulin-independent. Transplantation
resulted in stable blood glucose levels and the first seven patients
who received transplants "all remain insulin-free," Dr. Shapiro said.
"We've learned a couple of important lessons" from this series of
transplant patients, Dr. Shapiro told Reuters Health in an interview
during the meeting. First, the transplant team found that the
immunosuppressant tacrolimus, which they use post-transplantation,
has an adverse effect on kidney function in the few patients with
poor kidney function at the time of transplantation. Second,
immunosuppression has caused stomatitis and hyperlipidemia that has
been correctable with drug therapy.
"Islet cell transplantation is not for every patient," Dr. Shapiro
said. "It is for those patients with brittle disease who have wide
swings in blood glucose levels." However, in that subpopulation, the
procedure has proven to correct blood glucose levels and maintain
them at stable levels over the long-term, he said. He added that
there is little risk in the procedure. If it does not work, the
patient simply resumes insulin therapy. Dr. Shapiro noted that
advances are being made in other areas of transplantation, where
immunosuppression can be stopped after a finite period of time. He
hopes the same can be true with islet cell transplantation.
Multicenter trials of islet cell transplantation are set to start
soon.
UK charity launches diabetes transplant project By Patricia Reaney
LONDON 1-26-01 (Reuters) - A leading British charity launched a
research project on Friday which it hopes will lead to a cure for
diabetes. Diabetes UK said the programme is based on the work of
British-born Canadian surgeon Dr James Shapiro, who has perfected a
procedure to transplant insulin-producing islet cells into people
with diabetes. So far Shapiro and his team have transplanted islet
cells in 15 patients. Thirteen are now completely off insulin and the
remaining two are taking reduced doses.
"We have every reason to believe we can repeat the success of his
team," Moira Murphy, the director of research at Diabetes UK, told a
news conference. Islets are clusters of cells in the pancreas that
produce insulin, which regulates blood sugar levels. In people with
Type I diabetes their islets have been destroyed by their immune
system.
A woman in Michigan was the first person to undergo the experimental
cell transplant in 1998 when she received islet cells and bone marrow
cells from a deceased, unrelated donor.
Shapiro and his team at the University of Alberta in Edmonton, Canada
refined the islet transplant by using specific anti-rejection drugs
and islet cells from two donors. Diabetes UK will duplicate his
method in seven centres in Britain. "This is not a cure but it is a
major step forward," said Shapiro, who attended the launch.
Diabetes UK said it expects to do the first transplant in the summer
and to complete 10 within the first year of the project. The
transplant involves removing the cells from the donor pancreas,
purifying them and then injecting them into the liver of the patient
using a local anaesthetic and an X-ray imaging machine.
In Canada it has been dubbed a "drive-through transplant" because the
injection only takes 5-10 minutes and most patients are back to their
normal routine within 24 hours. But Shapiro emphasised it is not for
everyone. Only patients who have suffered frequent and dangerous
comas and early damage from diabetes will benefit. But they must take
anti-rejection drugs which carry an increase risk of cancer and
infection.
In Britain 1.4 million people have been diagnosed with diabetes and
an estimated one million more have it and don't know it. Type I
diabetes is the most serious form of the disease with patients
needing insulin injections It accounts for 10-25 percent of cases.
Type 2, or adult onset, is a milder condition that can be treated
with diet, exercise or drugs to stimulate the secretion of insulin.
Diabetes can cause kidney failure, strokes, heart attacks, blindness
and nerve damage. It affects 130 million people worldwide and kills
2.8 million each year. Experts estimate the number of sufferers will
increase to 220 million by the year 2010.

Islet-cell transplants show mixed results for diabetes

2007-11-24 22:18:27

http://seattletimes.nwsource.com/text/2001856196_isletcell12m.html
Islet-cell transplants show mixed results for diabetes
By Warren King
Seattle Times medical reporter
In the beginning, just after the transplant, Jackie Westcott didn't
have to worry as much about her delicate dance with diabetes.
The exhausting lows from too little blood sugar were not as common.
It was easier to keep the sugar, or glucose, on an even keel. She
didn't worry that an unscheduled slice of bread might make her need
an extra shot of insulin.
Westcott was elated with the initial results of a transplant of
islets, the clusters of pancreas cells that produce insulin and
regulate glucose. She was one of dozens of patients nationwide
receiving the experimental therapy scientists hoped would free
diabetics of insulin injections or, at the least, help them better
control their disease and prevent crippling complications.
But within months of the procedure, performed in Seattle, Westcott
was back where she started: needing just as much injected insulin as
before.
"I'm disappointed," said the 43-year-old Petersburg, Alaska,
resident, "but I'm not unhappy, because I'm hoping (the experiment)
is helping other diabetics out there."
Islet transplants, using cells from deceased donors, have been the
great hope for helping patients with type 1 diabetes, a genetic form
also known as juvenile diabetes that develops early in life and
affects about 1 million people in the United States. But the results
have been mixed.
Only about half the patients in the most-experienced transplant
centers no longer need insulin injections; the other half have better
glucose control, but many still need injections. Less-experienced
centers have had even less success.
In Seattle, three of six transplant patients are better able to
control their glucose levels, and two of those are likely to get off
insulin injections, said Dr. Paul Robe